Tom 15, Nr 3 (2020)
KARDIOLOGIA EKSPERYMENTALNA
Opublikowany online: 2020-12-10
Wyświetlenia strony
332
Wyświetlenia/pobrania artykułu
100
Redukcja pojedyńczej i złożonej arytmii komorowej w ostrej fazie zawału serca w przebiegu stosowania bezpośredniej hipotermii serca — badanie na modelu zwierzęcym
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, 2, 3, 4, 5
, 2, 3, 4, 5
Kardiol Inwazyjna 2020;15(3):154-157.
Streszczenie
Okołozawałowe arytmie komorowe mogą być przyczyną pogorszenia
stanu klinicznego pacjentów, czasami wymagają
przedłużonej hospitalizacji na oddziale intensywnego nadzoru
kardiologicznego, a także leczenia zabiegowego. Mechanizmem
prowadzącym do ich wystąpienia jest głównie zjawisko
re-entry, rzadziej dyspersja repolaryzacji. Farmakologiczne
i niefarmakologiczne postępowanie w przypadku takich arytmii
obejmuje stosowanie beta-adrenolityków, amiodaronu,
lignokainy, kardiowertera-defibrylatora, ablacji oraz modulacji
neuroosiowej w przypadku burzy elektrycznej. W prezentowanej
pracy przedstawiono wpływ bezpośredniej hipotermii serca
(DHH) na artmie komorowe towarzyszące ostrej fazie zawału
serca u świni.
Słowa kluczowe: zawał sercaczęstoskurcz komorowyprzedwczesne pobudzenia komorowebezpośrednia hipotermia serca
Referencje
- Kléber AG, Janse MJ, van Capelle FJ, et al. Mechanism and time course of S-T and T-Q segment changes during acute regional myocardial ischemia in the pig heart determined by extracellular and intracellular recordings. Circ Res. 1978; 42(5): 603–613.
- Janse M. Electrophysiological mechanisms of ventricular arrhythmias resulting from myocardial ischaemia and infarction. Physiol Rev. 1989; 69: 1049–1069.
- Allessie MA, Bonke FI, Schopman FJ. Circus movement in rabbit atrial muscle as a mechanism of tachycardia. III. The "leading circle" concept: a new model of circus movement in cardiac tissue without the involvement of an anatomical obstacle. Circ Res. 1977; 41(1): 9–18.
- Avezum A, Piegas LS, Goldberg RJ, et al. GRACE Investigators. Magnitude and prognosis associated with ventricular arrhythmias in patients hospitalized with acute coronary syndromes (from the GRACE Registry). Am J Cardiol. 2008; 102(12): 1577–1582.
- Bigger JT, Fleiss JL, Kleiger R, et al. The relationships among ventricular arrhythmias, left ventricular dysfunction, and mortality in the 2 years after myocardial infarction. Circulation. 1984; 69(2): 250–258.
- Funaro S, La Torre G, Madonna M, et al. AMICI Investigators. Incidence, determinants, and prognostic value of reverse left ventricular remodelling after primary percutaneous coronary intervention: results of the Acute Myocardial Infarction Contrast Imaging (AMICI) multicenter study. Eur Heart J. 2009; 30(5): 566–575.
- Priori SG, Blomström-Lundqvist C, Mazzanti A, et al. Task Force for the Management of Patients with Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death of the European Society of Cardiology (ESC). 2015 ESC Guidelines for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death: the task force for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death of the European Society of Cardiology (ESC) endorsed by: association for European Paediatric and Congenital Cardiology (AEPC). Eur Heart J. 2015; 36: 2793–2867.
- Antzelevitch Ch. Drug-induced spatial dispersion of repolarization Cardiol J. 2008; 15: 100–121.
- Arini PD, Baglivo FH, Martínez JP, et al. Evaluation of ventricular repolarization dispersion during acute myocardial ischemia: spatial and temporal ECG indices. Med Biol Eng Comput. 2014; 52(4): 375–391.
- Ziqing Y, Zhangwei Ch, Juan W, et al. Electrocardiographic parameters effectively predict ventricular tachycardia/fibrillation in acute phase and abnormal cardiac function in chronic phase of ST-segment elevation myocardial infarction. J Cardiovasc Electrophysiol. 2018; 29(5): 756–766.
- Zaidi M, Robert A, Fesler R, et al. Dispersion of ventricular repolarisation: a marker of ventricular arrhythmias in patients with previous myocardial infarction. Heart. 1997; 78(4): 371–375.
- Tofler GH, Stone PH, Muller JE, et al. Prognosis after cardiac arrest due to ventricular tachycardia or ventricular fibrillation associated with acute myocardial infarction (the MILIS Study). Multicenter Investigation of the Limitation of Infarct Size. Am J Cardiol. 1987; 60(10): 755–761.
- Newby KH, Thompson T, Stebbins A, et al. Sustained ventricular arrhythmias in patients receiving thrombolytic therapy: incidence and outcomes. The GUSTO Investigators. Circulation. 1998; 98(23): 2567–2573.
- Mehta RH, Starr AZ, Lopes RD, et al. APEX AMI Investigators. Incidence of and outcomes associated with ventricular tachycardia or fibrillation in patients undergoing primary percutaneous coronary intervention. JAMA. 2009; 301(17): 1779–1789.
- Bourke T, Vaseghi M, Michowitz Y, et al. Neuraxial Modulation for Refractory Ventricular Arrhythmias. Circulation. 2010; 121(21): 2255–2262.
- Varon J, Acosta P. Therapeutic hypothermia: past, present, and future. Chest. 2008; 133(5): 1267–1274.
- Polderman KH. Mechanisms of action, physiological effects, and complications of hypothermia. Crit Care Med. 2009; 37(7 Suppl): S186–S202.
- Steg PG, James SK, Atar D. ESC Guidelines for the management of acute myocardial infarction in patients presenting with ST-segment elevation.Task Force on the management of ST-segment elevation acute myocardial infarction of the European Society of Cardiology. Eur Heart J. 2012; 33(2569): 2619.
- Callaway CW, Donnino MW, Fink EL, et al. Part 8: Post-Cardiac Arrest Care: 2015 American Heart Association Guidelines Update for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Circulation. 2015; 132(18 Suppl 2): S465–S482.
- Hypothermia after Cardiac Arrest Study Group. Mild therapeutic hypothermia to improve the neurologic outcome after cardiac arrest. N Engl J Med. 2002; 346(8): 549–556.
- Bernard SA, Gray TW, Buist MD, et al. Treatment of comatose survivors of out-of-hospital cardiac arrest with induced hypothermia. N Engl J Med. 2002; 346(8): 557–563.
