open access

Vol 58, No 2 (2024)
Letter to the Editors
Submitted: 2023-10-29
Accepted: 2024-02-14
Published online: 2024-03-07
Get Citation

Two cases of delayed onset, fully reversible cortical oedema and signal intensity on brain MRI without infarction caused by prolonged migraine aura

Roaa Zayat1, Olga P. Fermo1
·
Pubmed: 38450724
·
Neurol Neurochir Pol 2024;58(2):210-214.
Affiliations
  1. Department of Neurology, Mayo Clinic, Jacksonville, Florida, United States

open access

Vol 58, No 2 (2024)
Letters to the Editors
Submitted: 2023-10-29
Accepted: 2024-02-14
Published online: 2024-03-07

Abstract

Not available

Abstract

Not available
Get Citation

Keywords

migraine, aura, magnetic resonance imaging, persistent, prolonged, headache, reversible, brain

About this article
Title

Two cases of delayed onset, fully reversible cortical oedema and signal intensity on brain MRI without infarction caused by prolonged migraine aura

Journal

Neurologia i Neurochirurgia Polska

Issue

Vol 58, No 2 (2024)

Article type

Letter to the Editors

Pages

210-214

Published online

2024-03-07

Page views

132

Article views/downloads

106

DOI

10.5603/pjnns.98031

Pubmed

38450724

Bibliographic record

Neurol Neurochir Pol 2024;58(2):210-214.

Keywords

migraine
aura
magnetic resonance imaging
persistent
prolonged
headache
reversible
brain

Authors

Roaa Zayat
Olga P. Fermo

References (12)
  1. Buse DC, Loder EW, Gorman JA, et al. Sex differences in the prevalence, symptoms, and associated features of migraine, probable migraine and other severe headache: results of the American Migraine Prevalence and Prevention (AMPP) Study. Headache. 2013; 53(8): 1278–1299.
  2. Headache Classification Committee of the International Headache Society (IHS). The International Classification of Headache Disorders, 3rd edition (beta version). Cephalalgia. 2013; 33(9): 629–808.
  3. Leao A. Further observations on the spreading depression of activity in the cerebral cortex. J Neurophysiol. 1947; 10(6): 409–414.
  4. Dreier JP. The role of spreading depression, spreading depolarization and spreading ischemia in neurological disease. Nat Med. 2011; 17(4): 439–447.
  5. Lauritzen M, Dreier JP, Fabricius M, et al. Clinical relevance of cortical spreading depression in neurological disorders: migraine, malignant stroke, subarachnoid and intracranial hemorrhage, and traumatic brain injury. J Cereb Blood Flow Metab. 2011; 31(1): 17–35.
  6. Resnick S, Reyes-Iglesias Y, Carreras R, et al. Migraine with aura associated with reversible MRI abnormalities. Neurology. 2006; 66(6): 946–947.
  7. Stuckey SL, Goh TD, Heffernan T, et al. Hyperintensity in the subarachnoid space on FLAIR MRI. AJR Am J Roentgenol. 2007; 189(4): 913–921.
  8. Bereczki D, Kollár J, Kozák N, et al. Cortical spreading edema in persistent visual migraine aura. Headache. 2008; 48(8): 1226–1229.
  9. Kim YJ, Kwon SU. Recurrent steroid-responsive cerebral vasogenic edema in status migrainosus and persistent aura. Cephalalgia. 2015; 35(8): 728–734.
  10. Smith M, Cros D, Sheen V. Hyperperfusion with vasogenic leakage by fMRI in migraine with prolonged aura. Neurology. 2002; 58(8): 1308–1310.
  11. Belvís R, Ramos R, Villa C, et al. Brain apparent water diffusion coefficient magnetic resonance image during a prolonged visual aura. Headache. 2010; 50(6): 1045–1049.
  12. Gómez-Choco M, Capurro S, Obach V. Migraine with aura associated with reversible sulcal hyperintensity in FLAIR. Neurology. 2008; 70(24 Pt 2): 2416–2418.

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