open access

Vol 46, No 3 (2012)
OPIS PRZYPADKU
Submitted: 2011-01-19
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Bilateral paramedian thalamic infarction with hypothalamic dysfunction

Ewa Papuć, Joanna Wojczal, Zbigniew Stelmasiak, Konrad Rejdak
DOI: 10.5114/ninp.2012.29135
·
Neurol Neurochir Pol 2012;46(3):279-283.

open access

Vol 46, No 3 (2012)
OPIS PRZYPADKU
Submitted: 2011-01-19

Abstract

Unilateral thalamic lesions cause transient or permanent behavioral, sensory and oculomotor disturbances; bilateral lesions of thalamus result in more severe and longer lasting symptoms. We present an atypical case of bilateral paramedian thalamic infarct with concomitant hypothalamic dysfunction. The only risk factor of ischaemic stroke found in the patient was a short lasting episode of atrial fibrillation. Bilateral paramedian thalamic infarcts may result from occlusion of one paramedian thalamic artery, which arises from the posterior cerebral artery, either with separated or with a common trunk, thus supplying the thalamus bilaterally. Independently of anatomical variants of thalamus blood supply, the most probable cause of infarct in our patient was unilateral or bilateral occlusion of the posterior cerebral artery by cardioembolism, probably in the course of basilar artery occlusion. Hypothalamic dysfunction may accompany thalamic infarcts; thus hypothalamo-pituitary function should be routinely assessed in bithalamic infarcts.

Abstract

Unilateral thalamic lesions cause transient or permanent behavioral, sensory and oculomotor disturbances; bilateral lesions of thalamus result in more severe and longer lasting symptoms. We present an atypical case of bilateral paramedian thalamic infarct with concomitant hypothalamic dysfunction. The only risk factor of ischaemic stroke found in the patient was a short lasting episode of atrial fibrillation. Bilateral paramedian thalamic infarcts may result from occlusion of one paramedian thalamic artery, which arises from the posterior cerebral artery, either with separated or with a common trunk, thus supplying the thalamus bilaterally. Independently of anatomical variants of thalamus blood supply, the most probable cause of infarct in our patient was unilateral or bilateral occlusion of the posterior cerebral artery by cardioembolism, probably in the course of basilar artery occlusion. Hypothalamic dysfunction may accompany thalamic infarcts; thus hypothalamo-pituitary function should be routinely assessed in bithalamic infarcts.

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Keywords

ischaemic stroke, bilateral thalamic infarct, hypothalamic dysfunction

About this article
Title

Bilateral paramedian thalamic infarction with hypothalamic dysfunction

Journal

Neurologia i Neurochirurgia Polska

Issue

Vol 46, No 3 (2012)

Pages

279-283

DOI

10.5114/ninp.2012.29135

Bibliographic record

Neurol Neurochir Pol 2012;46(3):279-283.

Keywords

ischaemic stroke
bilateral thalamic infarct
hypothalamic dysfunction

Authors

Ewa Papuć
Joanna Wojczal
Zbigniew Stelmasiak
Konrad Rejdak

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