Vol 5, No 5 (2009)
Review paper
Published online: 2009-12-01

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Continuing trastuzumab beyond progression in breast cancer patients - pros and cons

Sylwia Dębska, Piotr Potemski
Onkol. Prak. Klin 2009;5(5):189-197.

Abstract

Anti-HER2 monoclonal antibody trastuzumab is currently regarded as a standard therapy for patients with HER2-positive breast cancer. This drug is used in adjuvant treatment as well as in metastatic breast cancer. In palliative treatment, trastuzumab is effective both in monotherapy and in combination with chemotherapy. However, there is no standard therapeutic strategy for patients who have progressed after therapy with trastuzumab. It is strongly believed that continuing trastuzumab beyond progression is beneficial for patients. This opinion results from complex mechanism of the drug action and unknown way of drug resistance emergence. This paper is a short review of current literature concerning treatment strategy for patients with progression during trastuzumab-based therapy. Analyzed strategies are: continuing trastuzumab beyond progression with replacement of combined cytotoxic drug, stopping trastuzumab and starting consecutive chemotherapy, and starting treatment with lapatinib - oral inhibitor of epidermal growth factor receptor and HER2.

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References

  1. Ross JS, Slodkowska EA, Symmans WF, et al. The HER-2 receptor and breast cancer: ten years of targeted anti-HER-2 therapy and personalized medicine. Oncologist. 2009; 14(4): 320–368.
  2. Schechter AL, Stern DF, Vaidyanathan L, et al. The neu oncogene: an erb-B-related gene encoding a 185,000-Mr tumour antigen. Nature. 1984; 312(5994): 513–536.
  3. Dendukuri N, Khetani K, McIsaac M, et al. Testing for HER2-positive breast cancer: a systematic review and cost-effectiveness analysis. CMAJ. 2007; 176(10): 1429–1434.
  4. Ridolfi RL, Jamehdor MR, Arber JM. HER-2/neu testing in breast carcinoma: a combined immunohistochemical and fluorescence in situ hybridization approach. Mod Pathol. 2000; 13(8): 866–873.
  5. Musolino A, Naldi N, Bortesi B, et al. Immunoglobulin G fragment C receptor polymorphisms and clinical efficacy of trastuzumab-based therapy in patients with HER-2/neu-positive metastatic breast cancer. J Clin Oncol. 2008; 26(11): 1789–1796.
  6. Singer CF, Köstler WJ, Hudelist G. Predicting the efficacy of trastuzumab-based therapy in breast cancer: current standards and future strategies. Biochim Biophys Acta. 2008; 1786(2): 105–113.
  7. Nishimura R, Okumura Y, Arima N. Trastuzumab monotherapy versus combination therapy for treating recurrent breast cancer: time to progression and survival. Breast Cancer. 2008; 15(1): 57–64.
  8. Slamon DJ, Leyland-Jones B, Shak S, et al. Use of chemotherapy plus a monoclonal antibody against HER2 for metastatic breast cancer that overexpresses HER2. N Engl J Med. 2001; 344(11): 783–792.
  9. Marty M, Cognetti F, Maraninchi D, et al. Randomized phase II trial of the efficacy and safety of trastuzumab combined with docetaxel in patients with human epidermal growth factor receptor 2-positive metastatic breast cancer administered as first-line treatment: the M77001 study group. J Clin Oncol. 2005; 23(19): 4265–4274.
  10. Burstein HJ, Keshaviah A, Baron AD, et al. Trastuzumab plus vinorelbine or taxane chemotherapy for HER2-overexpressing metastatic breast cancer: the trastuzumab and vinorelbine or taxane study. Cancer. 2007; 110(5): 965–972.
  11. Gori S, Colozza M, Mosconi AM, et al. Phase II study of weekly paclitaxel and trastuzumab in anthracycline- and taxane-pretreated patients with HER2-overexpressing metastatic breast cancer. Br J Cancer. 2004; 90(1): 36–40.
  12. Montemurro F, Donadio M, Clavarezza M, et al. Outcome of patients with HER2-positive advanced breast cancer progressing during trastuzumab-based therapy. Oncologist. 2006; 11(4): 318–324.
  13. Cancello G, Montagna E, D'Agostino D, et al. Continuing trastuzumab beyond disease progression: outcomes analysis in patients with metastatic breast cancer. Breast Cancer Res. 2008; 10(4): R60.
  14. Bartsch R, Wenzel C, Altorjai G, et al. Capecitabine and trastuzumab in heavily pretreated metastatic breast cancer. J Clin Oncol. 2007; 25(25): 3853–3858.
  15. Bartsch R, Wenzel C, Gampenrieder SP, et al. Trastuzumab and gemcitabine as salvage therapy in heavily pre-treated patients with metastatic breast cancer. Cancer Chemother Pharmacol. 2008; 62(5): 903–910.
  16. Bartsch R, Wenzel C, Hussian D, et al. Analysis of trastuzumab and chemotherapy in advanced breast cancer after the failure of at least one earlier combination: an observational study. BMC Cancer. 2006; 6: 63.
  17. García-Sáenz JA, Martín M, Puente J, et al. Trastuzumab associated with successive cytotoxic therapies beyond disease progression in metastatic breast cancer. Clin Breast Cancer. 2005; 6(4): 325–329.
  18. THOR Study: A study of continued herceptin (Trastuzumab) in combination with second line chemotherapy in patients with HER2 positive metastatic breast cancer. http://clinicaltrials.gov/ct2/show/ /NCT00448279.
  19. A study of herceptin (Trastuzumab) in combination with 2nd-line chemotherapy in patients with HER2 positive metastatic breast cancer. http://clinicaltrials.gov/show/NCT00444587..
  20. Benefit of adding trastuzumab to second line chemotherapy in breast cancer patients previously treated with trastuzumab. http://www.clinicaltrials.gov/ct/show/NCT00130507.
  21. von Minckwitz G, du Bois A, Schmidt M, et al. Trastuzumab beyond progression in human epidermal growth factor receptor 2-positive advanced breast cancer: a german breast group 26/breast international group 03-05 study. J Clin Oncol. 2009; 27(12): 1999–2006.
  22. Geyer CE, Forster J, Lindquist D, et al. Lapatinib plus capecitabine for HER2-positive advanced breast cancer. N Engl J Med. 2006; 355(26): 2733–2743.
  23. Morabito A, Piccirillo MC, De Ma, et al. Trastuzumab beyond progression: is the risk of cardiac toxicity really not increased? J Clin Oncol. 2009; 27: e123.